Chest Email Content Delivery
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
QUICK SEARCH:   [advanced]


     

Guest Access | Sign In via User Name/Password
This Article
Right arrow Full Text (PDF) Free
Right arrow Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when eLetters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Article Archive
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Castellanos, A.
Right arrow Articles by Arcebal, A. G.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Castellanos, A., Jr.
Right arrow Articles by Arcebal, A. G.
(Chest. 1969;56:470-476.)
© 1969 American College of Chest Physicians

Mechanisms of Slow Ventricular Tachycardias in Acute Myocardial Infarction

Agustin Castellanos Jr. M.D.1; Louis Lemberg M.D., F.C.C.P.1; and Azucena G. Arcebal M.D.1

1 Section of Cardiology, Department of Medicine, University of Miami School of Medicine, The Division of Electrophysiology, Jackson Memorial Hospital and the Veterans Administration Hospital, Miami, Florida

Unifocal ventricular rhythms with rates between 60 and 100 per minute were classified as slow ventricular tachycardias when occurring in patients with acute myocardial infarction. Analysis of selected electrocardiograms showed that the paroxysms were usually automatic, nonparasystolic and intermittent. However, in one case, the arrhythmia resulted from the exit block of a faster, parasystolic, ventricular tachycardia. The first ectopic beat in each series appeared either as an escape or as a late extrasystole. Variations in rate attributed to a disturbance of automaticity and/or conductivity were frequently observed. The erratic and unpredictable behavior of this arrhythmia probably reflected the unstable condition of the injured cells during the early stages of myocardial infarction.






HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 1969 by the American College of Chest Physicians.