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(Chest. 1974;66:526-529.)
© 1974 American College of Chest Physicians

Atrial Tachycardia Secondary to Sino-Atrial Node Reentry

Zorel Paritzky M.D.1; Kanji Obayashi M.D.1; and William J. Mandel M.D.2

1 Department of Cardiology, Cedars-Sinai Medical Center and the Department of Medicine, University of California at Los Angeles
2 Milly Factor Clinical Investigator of the Western Cardiac Foundation

In one patient reproducible episodes of atrial tachycardia could be elicited via programmed atrial premature depolarizations. Evaluation of the electrophysiologic studies suggested that the most likely mechanism was sino-atrial reciprocation. Intravenous atropine produced a pronounced shortening of sino-atrial conduction time (200 msec, control; 70 msec, atropine) and sustained atrial tachycardia no longer could be elicited. These studies support the concept that sino-atrial reciprocation can be a mechanism responsible for atrial tacharrhythmias in man.

Submitted on January 16, 1974
Accepted on April 29, 1974






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Copyright © 1974 by the American College of Chest Physicians.