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Chest, Vol 75, 189-192, Copyright © 1979 by American College of Chest Physicians

ARTICLES

Cardiac dysfunction in a patient with familial hypokalemic periodic paralysis

LD Kramer, JP Cole, JC Messenger and MH Ellestad

A 19-year-old white man with familial hypokalemic periodic paralysis developed evidence of cardiac dysfunction during a episode of flaccid paralysis. This consisted of elevated total creatine phosphokinase (CPK), an increased myocardial fraction of CPK (myocardial band), alteration in the lactic dehydrogenase isoenzyme pattern, severe bradycardia, and evidence of left ventricular dysfunction. These findings, in conjunction with selected cases from the literature, suggest the possibility that cardiomyopathy may be a heretofore unrecognized complication of this disorder.


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