Chest ACCP Career Connection
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
 QUICK SEARCH:   [advanced]


     

Guest Access | Sign In via User Name/Password
This Article
Right arrow Full Text (PDF) Free
Right arrow Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when eLetters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Article Archive
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Zack, M. B.
Right arrow Articles by Palange, A. V.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Zack, M. B.
Right arrow Articles by Palange, A. V.

Chest, Vol 88, 669-675, Copyright © 1985 by American College of Chest Physicians


ARTICLES

Oxygen supplemented exercise of ventilatory and nonventilatory muscles in pulmonary rehabilitation

MB Zack and AV Palange

An outpatient pulmonary rehabilitation program was evaluated for evidence of patient improvement. All patients exercised on 4 LPM oxygen (O2). Inspiratory resistive loading (ventilatory muscle modality) was prescribed to achieve one-half of the patient's maximal inspiratory force at minute ventilation (VE) not greater than one-and-one half times resting VE. Walking (nonventilatory muscle exercise) was prescribed at work level requiring VE of 50 percent of maximum breathing capacity, if a ventilatory limit to exercise, or a work level set at 60 percent of VO2 maximum, if no ventilatory limit to exercise. Significant improvement was noted after rehabilitation in maximum workload, 12 min walk, and endurance both on room air (RA) and O2, as compared to pre-rehabilitation values. No improvement was noted in resting pulmonary function, gas exchange, exercise-induced hypoxemia or VO2 max. O2 increased work performance compared to values in the same patients on RA, both before and after rehabilitation, an effect possibly mediated by O2-induced reduction in submaximal VE.





HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 1985 by the American College of Chest Physicians.