Chest ACCP Member Benefits
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
 QUICK SEARCH:   [advanced]


     

Guest Access | Sign In via User Name/Password
This Article
Right arrow Full Text (PDF) Free
Right arrow Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when eLetters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Article Archive
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Rizzato, G
Right arrow Articles by Sabbioni, E
Right arrow Search for Related Content
PubMed
Right arrow Articles by Rizzato, G
Right arrow Articles by Sabbioni, E

Chest, Vol 90, 101-106, Copyright © 1986 by American College of Chest Physicians


ARTICLES

Trace of metal exposure in hard metal lung disease

G Rizzato, S Lo Cicero, M Barberis, M Torre, R Pietra and E Sabbioni

A male subject exposed for many years to metal dusts from grinding and cutting hard metals was investigated four years after the exposure ceased. While chest x-ray films and histologic examinations showed diffuse interstitial fibrosis and severe perivascular and peribronchiolar fibrosis, radiochemical neutron activation analysis (NAA) showed high W, Ta and Co concentrations in the pulmonary specimen compared to the corresponding determinations in 17 control subjects. Tantalum was also found in high concentrations in bronchoalveolar lavage fluid (BAL) and in the blood. In addition other elements such as Ni, Cr, Th and Cd were found in the lung biopsy specimen of the worker in amounts significantly higher than in the control subjects. The results suggest that hard metal origin of pulmonary fibrosis can be detected many years after removal from exposure.


This article has been cited by other articles:


Home page
Am. J. Physiol. Lung Cell. Mol. Physiol.Home page
J. Araya, M. Maruyama, A. Inoue, T. Fujita, J. Kawahara, K. Sassa, R. Hayashi, Y. Kawagishi, N. Yamashita, E. Sugiyama, et al.
Inhibition of proteasome activity is involved in cobalt-induced apoptosis of human alveolar macrophages
Am J Physiol Lung Cell Mol Physiol, October 1, 2002; 283(4): L849 - L858.
[Abstract] [Full Text] [PDF]


Home page
Toxicol Ind HealthHome page
D. Cherdwongcharoensuk, E. M Cunha, S. Upatham, A. S. Pereira, M. J. R Oliveira, and A. P Aguas
In vivo ingestion of heavy metal particles of Se, Hg and W by murine macrophages. A study using scanning electron microscopy coupled with X-ray microanalysis
Toxicology and Industrial Health, September 1, 2002; 18(8): 397 - 403.
[Abstract] [PDF]




HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 1986 by the American College of Chest Physicians.