Chest Email Content Delivery
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
 QUICK SEARCH:   [advanced]


     

Guest Access | Sign In via User Name/Password
This Article
Right arrow Full Text (PDF) Free
Right arrow Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when eLetters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Article Archive
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Lorch, D.
Right arrow Articles by Sahn, S.
Right arrow Search for Related Content
PubMed
Right arrow Articles by Lorch, D.
Right arrow Articles by Sahn, S.

Chest, Vol 90, 802-805, Copyright © 1986 by American College of Chest Physicians


ARTICLES

Post-extubation pulmonary edema following anesthesia induced by upper airway obstruction. Are certain patients at increased risk?

DG Lorch and SA Sahn

Pulmonary edema due to upper airway obstruction can be observed in a variety of clinical situations. The predominant mechanism is increased negative intrathoracic pressure, although hypoxia and cardiac and neurologic factors may contribute. Laryngospasm associated with intubation and general anesthesia is a common cause of pulmonary edema in children. However, only seven cases of pulmonary edema presumably due to laryngospasm have been reported in adolescents and adults. Five of the seven had other risk factors for upper airway obstruction, and in four, the diagnosis of "laryngospasm" could be explained by other factors. Patients with underlying risk factors for upper airway obstruction, such as a forme fruste of sleep apnea or nasopharyngeal abnormalities, appear to be at increased risk for the development of pulmonary edema in the setting of intubation and anesthesia. This form of pulmonary edema usually resolves rapidly without the need for aggressive therapy or invasive monitoring.


This article has been cited by other articles:


Home page
Arch Facial Plast SurgHome page
R. Westreich, I. Sampson, C. M. Shaari, and W. Lawson
Negative-Pressure Pulmonary Edema After Routine Septorhinoplasty: Discussion of Pathophysiology, Treatment, and Prevention
Arch Facial Plast Surg, January 1, 2006; 8(1): 8 - 15.
[Abstract] [Full Text] [PDF]


Home page
ChestHome page
G. L. Ackland and M. G. Mythen
Negative Pressure Pulmonary Edema as an Unsuspected Imitator of Acute Lung Injury/ARDS
Chest, May 1, 2005; 127(5): 1867 - 1868.
[Full Text] [PDF]




HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 1986 by the American College of Chest Physicians.