Chest ACCP Education Calendar
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
QUICK SEARCH:   [advanced]


     

Guest Access | Sign In via User Name/Password
This Article
Right arrow Full Text (PDF) Free
Right arrow Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when eLetters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Article Archive
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Kohama, A
Right arrow Articles by Kamada, T
Right arrow Search for Related Content
PubMed
Right arrow Articles by Kohama, A
Right arrow Articles by Kamada, T

Chest, Vol 98, 794-800, Copyright © 1990 by American College of Chest Physicians

ARTICLES

Pathologic involvement of the left ventricle in chronic cor pulmonale

A Kohama, J Tanouchi, M Hori, A Kitabatake and T Kamada
First Department of Internal Medicine, Osaka University School of Medicine, Japan.

To determine whether or not the left ventricle is pathologically involved in patients with chronic cor pulmonale, right and left ventricular weights, wall thickness, myocyte diameters, and percentage of fibrosis in 18 autopsied hearts were examined in patients with chronic pulmonary disease (CPD); ten had right ventricular hypertrophy on their electrocardiograms, and eight were without right ventricular hypertrophy. Five with extracardiopulmonary disease were used as controls. The weight of the right ventricle was significantly increased in CPD when compared to control subjects. Walls of both ventricles were significantly thicker in CPD. Myocyte diameters of both ventricles were significantly greater in CPD. The percentage of fibrosis in the right ventricle was significantly greater in CPD. The percentage of fibrosis in the left ventricle was significantly greater only in patients with right ventricular hypertrophy. We concluded that the left ventricle was also involved pathologically in patients with chronic cor pulmonale in the end stage of the disease.


This article has been cited by other articles:


Home page
 Eur Heart JHome page
F. H. Rutten, M.-J. M. Cramer, D. E. Grobbee, A. P.E. Sachs, J. H. Kirkels, J.-W. J. Lammers, and A. W. Hoes
Unrecognized heart failure in elderly patients with stable chronic obstructive pulmonary disease
Eur. Heart J., September 2, 2005; 26(18): 1887 - 1894.
[Abstract] [Full Text] [PDF]

Home page
 ChestHome page
A. Vonk-Noordegraaf, J. T. Marcus, S. Holverda, B. Roseboom, and P. E. Postmus
Early Changes of Cardiac Structure and Function in COPD Patients With Mild Hypoxemia
Chest, June 1, 2005; 127(6): 1898 - 1903.
[Abstract] [Full Text] [PDF]

Home page
 Asian Cardiovasc. Thorac. Ann.Home page
W. T Vigneswaran, E. V Chomka, V. Jelnin, M J. Hernan, and F. J Podbielski
Cardiac Morphology in Lung Volume Reduction Surgery for Endstage Emphysema
Asian Cardiovasc Thorac Ann, March 1, 2003; 11(1): 48 - 51.
[Abstract] [Full Text] [PDF]

Home page
 Am. J. Respir. Crit. Care Med.Home page
S. M. Scharf, M. Iqbal, C. Keller, G. Criner, S. Lee, and H. E. Fessler
Hemodynamic Characterization of Patients with Severe Emphysema
Am. J. Respir. Crit. Care Med., August 1, 2002; 166(3): 314 - 322.
[Abstract] [Full Text] [PDF]


HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 1990 by the American College of Chest Physicians.