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* From the ACCP-SEEK program, reprinted with permission. Items are selected by Department Editors Richard S. Irwin, MD, FCCP, and John G. Weg, MD, FCCP. For additional information, phone 1-847-498-1400.
Correspondence to: Joseph E. Parrillo, MD, FCCP, Cooper University Hospital, One Cooper Plaza, Suite 404, Camden, NJ 08103; e-mail: Parrillo-Joseph{at}cooperhealth.edu
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This morning he awoke with substernal chest pressure that lasted < 10 min. Although the pain was similar to that of his myocardial infarction, he graded it only 3 on a scale of 10 in intensity. The patient came to the emergency department, where he was pain free. Physical examination revealed a pulse of 70 beats/min and irregular, and a BP of 130/80 mm Hg. The rest of his physical examination was normal. His ECG is shown (Fig 1 ). Blood is drawn for laboratory tests, and he is transferred to the ICU. Initial management of this patient should include which of the following?
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| Answer: C. Ventricular pacing wire |
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In Mobitz II block, the prognosis is poor. Many of these patients acquire higher degrees of atrioventricular (AV) block and syncope (Adams-Stokes attacks) due to progressive conduction disease. Mobitz type I, or Wenckebach block, has a better prognosis and will frequently not progress to higher degrees of AV block.
In the setting of acute myocardial infarction (probably not present in this patient), type I AV block usually accompanies inferior infarction, is transient, usually does not require temporary pacing, and usually responds to atropine. Type II block most commonly occurs in the setting of anterior infarction, can require temporary or permanent pacing, and is associated with a high mortality, usually due to pump failure. Anatomically, type I block usually occurs due to dysfunction in the AV node, whereas type II block represents damage to the His-Purkinje system.
In this patient with Mobitz type II second-degree AV block, a temporary and probably permanent ventricular (or AV) pacing wire is indicated because of the high likelihood of further progression of his AV block. His chest pain may have resulted from a slower heart rate due to onset of AV block, or the pain may represent unstable angina. The patients cardiac enzymes were normal, and a permanent AV pacemaker was placed.
Lidocaine, atropine, and dobutamine will not speed up the rate in patients with Mobitz type II AV block. There is no indication for an intra-aortic balloon pump, which is usually employed in cardiogenic shock.
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Castellanos, A, Cox, MM, Fernandez, PR, et al Mechanisms and dynamics of episodes of progression of 2:1 atrioventricular block in patients with documented 2-level conduction disturbances. Am J Cardiol 1992;71,193-199[CrossRef]
Gonzalez, MD, Scherlag, BJ, Mabo, P, et al Functional dissociation of cellular activation as a mechanism of Mobitz type II atrioventricular block. Circulation 1993;87,1389-1398
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F. J. Munoz and B. Thomas Mobitz Type I Block Chest, December 1, 2004; 126(6): 2025 - 2025. [Full Text] [PDF] |
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